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DenpaNews : Life After Coronavirus

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DenpaNews.com : Life After Coronavirus

Denpa News Even if you easily passed this viral test, there is no guarantee that your body has not suffered a serious blow and serious damage has been done to it.

Coronavirus, which turned with such force upside down the lives of billions of people around the world, really good known. Regardless, it’s still a big mystery. We open these secrets of SARS-Cov-2 and explain why each of us reacts to them differently. It’s a bit like a universe with a coronavirus or a virus in general. Scientists say the universe is infinite and growing, but for most of us the main question is what is behind this universe? The same with the virus. Is not living organism. Does that mean he’s dead? If he’s dead, you can’t kill? Then why is it even dangerous if he’s dead?

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The coronavirus is dead, but it’s not dead. Frankly speaking, the line between a living and non-living organism was established by scientists. They decided that a living organism should have a cell nucleus. Viruses don’t. V in a nutshell, they themselves are a simplified nucleus, without a cell. At first sight, it is a very primitive form. To protect the contents of the genetic material wrapped in a small amount of protein and a layer of fat. On this outer layer there are special groups of amino acids that form peculiar spikes, spikes. Therefore, in the photographs it is presented in the form of a ball covered with spikes. The flat image resembles a head with a crown. Hence the name: coronavirus.

The Power of Simplicity SARS-Cov-2

Why is it hard to kill the coronavirus? Because it’s so primitive. Bacterial infections are easier to fight. Even the simplest bacteria have to eat something. She needs food in order to generate energy that supports the entire structure. Eating is only half the battle, more chemicals are needed, often taken from the environment, such as enzymes, to distribute the food taken. Many of these chemicals are involved in the functioning of bacteria. Why is it so important? Because it is also the weakness of the microorganism. Scientists can look for substances characteristic of bacteria and then test how they are absorbed by our opponent. So they can deprive him of food or poison him. With viruses, everything is much more complicated. In most cases, they remain almost completely inactive. We already wrote that they are dead, but from time to time they are activated (more on that at some point). Thus, as you can see, viruses not only have a simple structure, but also function in the most simplified way. This brilliance of genetic material is striking in other ways as well. To understand why you must return to living organisms again.

Weaknesses of the Coronavirus

The simplest bacterium (Mycoplasma genitalium) has about 577 genes (that is, its entire genome). We got to know the mouse genome better in 2009 thanks to $100 million in research. It turned out that we have about 75% of the same genes as the mouse. It is estimated that we also have a total number of genes with it (about 43,162). It is important? Yes! Each gene can encode one protein. It is also an opportunity to find weaknesses in these proteins. So how many genes does the SARS-Cov-2 coronavirus have? About 10! How do we know this? In Wuhan, where the pandemic began, scientists have carefully studied it.

On January 17, the complete genome record was released at the National Center for Biotechnology Information (NIH) in Bethesda. It contains 29903 nucleotides. What’s the difference? 3 nucleotides (letters in the notation) is a codeon, or amino acid. These, in turn, are proteins. It’s hard to imagine, but there can be several thousand amino acids in one protein, but methionine is always the first. This is the one that allows scientists to extract individual proteins. So we also know that there are probably about 10 different proteins in a SARS-COV-2 particle. So that we can scream: we got him! Nothing like the coronavirus that caused Covid-19 is still a big mystery.

We know well how it behaves in the human body, but we do not know why this happens and what the various proteins are responsible for. Therefore, researchers take different scenarios based on previously known viruses and try to use them to find weaknesses in the structure of the killer responsible for the pandemic.

Description of the culprit of the pandemic

DenpaNews : Okay. We already know so much about it, so why is there no cure? We have already answered a little. A drug is a substance that acts on the opponent’s weaknesses. He cuts off his food, penetrates the enemy structure and destroys him from the inside, mobilizes allies from the immune system to fight. But first it must somehow reach him. If we were playing coronavirus tracking detector, we would first need a description to locate it, floating in the exhaled air of an infected person, or hiding somewhere on a doorknob in a store.

This description might look like this: at first glance, this is an incredibly small, round dot about 0.3 microns in size. If we track it on the handle, we’ll only see these dots with the laces. It is best to see there that they are not living organisms. They just float in liquid. If they are on this handle, they will lose their ability to activate for too long. We would say they would die, but they are dead, so that would be inaccurate. The time they can be out of the human body may vary depending on temperature or level ground. The longest because about 72 hours will survive on plastic and stainless surfaces. On the box – 24 hours, it can float up to 3 hours in the air.

Effective weapon against coronavirus

So we came to the first effective weapon against coronavirus. Our body has two types of immunity: specific (congenital) and nonspecific (acquired). Among congenital protective systems, the first barrier is passive innate immunity, i.e. skin and mucous membranes. We already know that the coronavirus can lurk on street for a certain amount of time. That’s why it’s so important that he doesn’t break through first obstacles. The mask is supposed to limit the possibility entry of SARS-Cov-2 into the body through the nose or mouth. Washing their hands disinfecting or wearing gloves also really keeps them out of your mouth and nose. This is because it is not able to penetrate the body through the skin. Touching with fingers to the area of ​​the face with viruses on the surface increases the risk of getting sick.

However, we have not yet explained why Coronavirus, roughly speaking, is dead and can be so dangerous for us. Exactly because it’s so primitive. In a nutshell, this little genetic the material contains the main purpose of the virus – reproduction. For this he needs a cell, in the case of the SARS-Cov-19 coronavirus, it is a human cell.
The entire structure of the virus is oriented towards adherence to the cell (hence, for example, protrusions), getting inside her and releasing her genetic material (RNA). The RNA then replicates (multiplies) and then the envelope is formed and released from the cell in large quantities to find another place to replication.
A spike in the coronavirus. Fortunately, the new coronavirus does not attack every cell. V at the end of the spike is a specific amino acid called “S” (from the word “thorn”). Researchers discovered that it is a molecule that only recognizes one type of another a substance found in a specific ACE enzyme. This enzyme converts other substance: angiotensin from form I to II. Angiotensin II (ACE2) is found in the lungs, kidneys, heart and intestines. To sum it up: SARS-Cov-19 coronavirus must go a long way and miss many protective measures. Only when in contact with ACE2 does it become truly dangerous. That’s when he starts doing what it can do best: multiply. With the help of the “S” protein, he convinces the victim (ACE2) to let him in. This is where the process starts multiplication, which can take up to 14 days. Therefore, the quarantine lasts 14 days, because during this time there may be no symptoms of the disease. The virus sits quietly within the cell and shares its genetic material.

But our body is not defenseless. We have already written about first line, innate immunity. Now all mechanisms can join the battle. Among the cells of the immune system, we single out, for example, phagocytes (to which include macrophages and granulocytes). These are terrible sharks. Their main purpose in of life – ingestion of pathogens (i.e. substances that would be considered threatening, therefore, the coronavirus becomes another pathogen for them). However, in order to for such a glutton to come down to the feast, she must find out that nearby have food. It may be notified by its own receptors or by substances called cytokines.

All hope for our immunity

DenpaNews : This brings us to the heart of the problem of fighting the coronavirus. If our immune system is very efficient, it quickly recognizes the threat, quickly releases a lot of glands. This coronavirus will die before it becomes a human. That is why the course of the disease can vary in intensity.

When we choke out SARS-Cov-2 at the very beginning, we may not even notice the symptoms of this fight. The more viruses multiply, the wider the battlefield, the more inflammatory substances (pro-inflammatory cytokines) appear, which should mobilize more resources of the immune system, T, B, NK cells. A war in the lungs where ACE2 gets more serious. Where the wood is crushed, chips fly, and in the case of coronavirus, a large amount of liquid is formed. The more opponents, the more fluid begins to fill the lungs. This is why patients complain about breathing problems. Weakened immunity can begin to be lost. The lungs fill with thick fluid, and in the worst case, so much fluid that you suffocate and die.

Of course, this is a very simplified military relationship, but it shows the importance of the effectiveness of our immune system and the performance of the whole organism.

Why are we responding differently to the coronavirus?

We each have our own ability to protect ourselves. This is why we react differently to the coronavirus. According to researchers, (statistically) children have the strongest immune systems. As we age, it may weaken. In addition, there are other diseases that “benefit” from having a defensive blade aimed at SARS-Cov-2. Hence the reports of death from comorbidities.

Coronavirus medicine

DenpaNews : Many different drugs are currently being tested, which are related to other military strategies. Some doctors try to strengthen the immune system so it can fight. Others are looking for a substance that can interrupt the replication process in ACE2 cells. Efforts are being made to block the “S” protein, or to give the sick immune system cells to those who have already won the battle and carry specialized substances against SARS-Cov-2. However, we still have many different disease scenarios depending on many internal factors. The primitive virus turns out to be an adversary with great survival power. Paradoxically, some researchers see this as an opportunity.

Viruses mutate

In the process of replication, minor changes in the structure of RNA can occur. This structure is not yet fully understood as “learning” to survive. That’s why there are viral diseases known for hundreds of years. The less lethal the disease, the more likely it is to “get along” with humans in some way. The deadliest viruses cause relatively short-range epidemics. They kill carriers too quickly to infect others. Thus, mutations can lead to a decrease in the ability to kill in order to survive longer. This is the talent that SARS-Cov-2 has. The long incubation time makes it possible to move into more cells before they die. The new coronavirus already knows this. But it kills too much for the entire population of this virus to survive. There is a chance that the mutation will reduce the strength of the human body. We know SARS can change. He managed to mutate from an animal virus to a human. The problem is that geneticists do not know to what extent changes in just 10 genes will occur … It is up to us to strengthen immunity, wash our hands, wear masks and wait for a cure or vaccine.

Life after COVID 19

DenpaNews : Coronovirus remains the most insidious disease known to history epidemics. And its insidiousness lies in the unpredictability of the consequences during the period recovery. Even if you are easy overcame this viral challenge, no guarantee that your body has not endured a serious blow and serious damage. However, the greatest risks of development delayed complications occur in  patients who have had coronovirus in moderate and severe forms.

Physicians have already accumulated sufficient experience and understanding the mechanism of harmful effects on the human body virus Covid 19. First of all, the pulmonary system suffers, which inevitably leads to a violation oxygen exchange in body tissues. This in turn starts a chain reaction formation of thrombosis and leads to a serious functional pathology of the internal organs.

For to assess the severity of the viral strike, in a network of clinics “Ramsey-Diagnostics” has developed special “Programs for post-COVID-19 diagnostics”, which allow the most reliable assessment of the degree of the applied virus damage and the risk of developing possible pathologies. “Post-Covid Programs diagnostics” in Ramsey allow you to include both laboratory tests and instrumental CT diagnostics, which allows assess the risks of developing pathologies and determine a further treatment strategy and recovery.

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